Our findings broadly establish companion dogs as a translational model for uncovering molecular mechanisms of lifespan variation, epigenomic instability, and age-related disease. We demonstrate that shorter-lived dogs do not merely die earlier; they undergo accelerated molecular aging specific to genomic locations and structures. Thus, lifespan variation within a species is reflected not only in the pace of epigenetic aging but also in the genomic architecture through which aging unfolds.

Epigenetic aging and transposon dysregulation reflect size-related lifespan compression in dogs – https://www.science.org/doi/10.1126/science.aeb2986

Canine mortality. Kind of fits with this blog theme, no?